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Its varied potential mechanisms are thought to include interaction with inducible and/or endothelial nitric oxide-generating systems, by influencing serotonin, dopamine, GABA, and/or opioid metabolism, and/or by directly aiding blood flow, repair, regrowth, angiogenesis, and remodeling

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BPC-157 has been shown to: Increase vascular endothelial growth factor receptor 2 (VEGFR2) expression