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While the food addiction concept remains debated, neuroimaging studies have documented striking parallels between obese individuals and people with substance use disorders: Both conditions show reduced dopamine D2 receptor availability in the striatum (downregulation from chronic overstimulation) Both show altered connectivity between the prefrontal cortex and limbic regions (impaired top-down control over reward-driven behavior) Both show enhanced neural reactivity to drug/food cues in the amygdala and orbitofrontal cortex Both show reduced gray matter volume in prefrontal cortical regions involved in executive function and impulse control GLP-1R agonists may address the common neurocircuitry underlying both conditions by reducing dopaminergic signaling in the VTA-NAc pathway, effectively decreasing the reward salience of both food and addictive substances
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